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“Background Campylobacter jejuni (C. jejuni), a microaerophilic, spiral-shaped, flagellated Gram-negative bacterium, is the most frequent cause of human gastroenteritis worldwide [1]. C. jejuni infections are often caused by consumption of undercooked poultry, unpasteurised milk or contaminated water

[2]. Adhesion of C. jejuni to host cells plays an important role in colonisation of chickens and in human infection [3]. Campylobacter binding to host cell receptors is not mediated by fimbria or pili, like in E. coli and Salmonella[4]. As noted in a recent review, other bacterial cell structures may contribute to interaction of Campylobacter with host cells [5]. In some cases, bacterial adhesion can be mediated by oligosaccharides present on the surface of host cells [6, 7]. In other cases, it is a pathogen oligosaccharide that is responsible for binding to specific, lectin-like, host cell structures. For example, a pathogenic Gram-positive bacterial species Nocardia rubra binds to a human lectin (intelectin) expressed by cells in different organs including intestine [8].