18 +/- 7 92 and 10 99 +/- 8 98 fT/cm,

respectively, and t

18 +/- 7.92 and 10.99 +/- 8.98 fT/cm,

respectively, and the mean laterality index, expressed by [(left - right)/(left + right)] was 0.025 +/- 0.104. This parameter can be useful for evaluating patients with unilateral sensory abnormality of the tongue. (C) 2008 Elsevier Ireland Ltd and the Japan Neuroscience Society. All rights reserved.”
“Recent studies have suggested that neuronal apoptosis in cerebral ischemia could arise from dysfunction of endoplasmic reticulum (ER) and mitochondria. B-cell lymphoma/leukemia-2 gene (Bcl-2) has been described as an inhibitor Selleckchem Flavopiridol both in programmed cell death (PCD) and ER dysfunction during apoptosis, and the Bcl-2 family play a key role in regulating the PCD, both locally at the ER and from a distance at the mitochondrial membrane. However, its signal pathways and concrete mechanisms in endoplasmic reticulum-initiated apoptosis remain incompletely understood. We therefore

investigate whether ischemia/reperfusion (I[R) selleck kinase inhibitor causes neuronal apoptosis in part via cross-talk between ER and mitochondria or not, and how the overexpression of Bcl-2 prevents this form of cell death. Here we show that analogous I/R-induced cell death occurs consequent to interactions of ER stress and mitochondrial death pathways. The participation of the mitochondrial pathway was demonstrated by the release of cytochrome C (cyt C) from mitochondrial into cytoplasmic fractions and caspase-9 cleavage. The involvement of ER stress was further supported by the observable increase of glucose-regulated protein 78(GRP78)/BiP expression and caspase-12 activity. Furthermore, prior to these changes, swelling of the ER lumen and dissociation of ribosomes from rough ER were detected by electron microscopy. Bcl-2 overexpression inhibits the release of SB-3CT cyt C and the activation of caspase-9/-8/-3 but not caspase-12 based on the results of Western blot. These suggest that cross-talk

between ER and mitochondria participate in neuronal damage after ischemia/reperfusion. Bcl-2 overexpression could suppress I/R-induced neuronal apoptosis via influencing mitochondrial integrity. (C) 2008 Elsevier Ireland Ltd and the Japan Neuroscience Society. All rights reserved.”
“Background In rural India, most births take place in the home, where high-risk care practices are common. We developed an intervention of behaviour change management, with a focus on prevention of hypothermia, aimed at modifying practices and reducing neonatal mortality.

Methods We did a cluster-randomised controlled efficacy trial in Shivgarh, a rural area in Uttar Pradesh.

Kidney International (2011) 79, 966-976; doi:10 1038/ki 2010 535;

Kidney International (2011) 79, 966-976; doi:10.1038/ki.2010.535; published online 19 January 2011″
“High-density ERPs evoked by rotated alphanumeric characters were examined to determine how neural processing is affected by stimulus orientation during letter/digit classifications and during mirror/normal discriminations. The former task typically produces response times that are unaffected by stimulus orientation while

the latter is thought to require mental rotation. Sensitivity to orientation was first observed around 100-140 ms and this effect was attributed to differences in low-level features between vertical and oblique orientations. Subsequently, character misorientation amplified the N170, a neural marker of object classification, between 160 and 220 ms. Top-down processing is reflected in the ERPs beginning at 280-320 ms and this GW3965 cost time range may reflect binding of ventral and dorsal stream information. In the case of mirror-normal discrimination these top-down

processes can lead to mental rotation between 340 and 700 ms. Therefore, although neural processing reflects object orientation, these effects do not translate into increases in reaction-times PARP inhibitor or impaired accuracy for categorisation, and precede those that do in the mental-rotation task. (C) 2011 Elsevier Ltd. All rights reserved.”
“Foxp3(+) T-regulatory cells (Tregs) may suppress pathogenic inflammation; however, although transferred Tregs lessen glomerulonephritis in mice, the role of endogenous foxp3(+) cells is not known. To study this, we characterized endogenous foxp3(+) cells in accelerated anti-glomerular basement membrane (GBM) nephritis by using foxp3(GFP) reporter mice to track their responses in early and established disease. Further, diphtheria toxin was used to ablate

foxp3(+) Tregs in foxp3(DTR) mice after establishing an immune response. In this model, mice were immunized with sheep globulin in adjuvant, and sheep anti-mouse GBM globulin was injected after 4 days to initiate progressive histological and functional injury. Intrarenal leukocytic infiltrates were increased by day 3 but intrarenal foxp3(+) Tregs, present in interstitial and periglomerular areas, were only increased at day 7. Ablation of foxp3(+) Tregs after injection Nitroxoline of anti-GBM globulin increased renal injury and systemic T-cell responses, including increased interferon-gamma and interleukin-17A (IL-17A) production, but no change in antibody titers. Compared with foxp3(+) Tregs isolated from naive mice, those from immunized mice produced more IL-10 and more effectively regulated CD4(+)foxp3(-) responder T cells. Thus, endogenous foxp3(+) Tregs infiltrate the kidney in glomerulonephritis, and deleting foxp3(+) cells after the induction of immune responses upregulated T-cell reactions and enhanced disease. Hence, endogenous foxp3(+) cells have increased suppressive capacity after immune stimuli. Kidney International (2011) 79, 977-986; doi:10.1038/ki.2010.

4% of sites, infusion stopped itself at 12 2%, patient voided at

4% of sites, infusion stopped itself at 12.2%, patient voided at 61.0%, volume attained age expected capacity at 12.2%, the patient was uncomfortable at 4.9% and results varied at 7.3%.

Conclusions: Data reveal that voiding

cystourethrogram is performed differently across North America and no standard protocol exists for the procedure. These differences could significantly impact voiding cystourethrogram results among institutions and taint our ability to compare results in the literature.”
“In normal development and homeostasis and in many disease states, cells and tissues must overcome the challenge of oxygen deprivation (hypoxia). The nematode C. elegans is emerging as an increasingly powerful Selisistat price system in which to understand GSK3326595 clinical trial how animals adapt to moderate hypoxia and survive extreme hypoxic insults. This review provides an overview of C. elegans responses to hypoxia, ranging from adaptation and arrest to death, and highlights some of the recent studies that have provided important insights into hypoxia signaling and resistance. Many of the key genes and pathways are evolutionarily conserved, and C. elegans hypoxia research promises to inform our

understanding of oxygen-sensitive signaling and survival in mammalian development and disease.”
“Interleukin-10 (IL-10) has important anti-inflammatory effects and can be protective in inflammatory conditions, such as chronic pain and infection. Exploring factors that modulate IL-10 levels may provide insight into pathomechanisms of inflammatory conditions and may provide a method of neuroprotection during these conditions. Lipopolysaccharide (LPS) stimulation of astrocytes is a Non-specific serine/threonine protein kinase source of IL-10; hence, it is of interest to investigate factors that modulate this process. Glutamate is present in increased concentrations in inflammatory conditions, and astrocytes also express glutamate receptors. The present study, therefore, investigated whether glutamate modulates LPS stimulation of IL-10 release from neonatal spinal cord astrocytes. Enzyme-linked immunosorbent

assays (ELISAs) were used to quantify IL-10 release from cultured neonatal spinal cord astrocytes, and reverse transcriptase-polymerase chain reaction (RT-PCR) was used to measure IL-10 mRNA expression. Glutamate (1 mM) significantly increased LPS (1 mu g/ml)-stimulated IL-10 release from astrocytes by 166% and significantly upregulated IL-10 mRNA levels. Glutamate synergistically signaled through metabotropic glutamate receptor subgroups and the phospholipase C signaling pathway. Spinal cord astrocytes may, therefore, play a larger anti-inflammatory role than first thought in situations where glutamate and a high concentration of Toll-like receptor 4 (TLR4) agonists are present. (C) 2012 IBRO. Published by Elsevier Ltd. All rights reserved.

Microinfusions of ATO into the shell, but not the core, sub-regio

Microinfusions of ATO into the shell, but not the core, sub-region of the nucleus accumbens (NAcb) significantly decreased premature responding whereas infusions of MPH in the core, but not the shell, sub-region

significantly increased premature responding. However, neither ATO nor MPH significantly altered impulsive behavior when infused into the prelimbic or infralimbic cortices. The opposing effects of ATO and MPH in the NAcb core and shell on impulsivity were unlikely mediated by ancillary effects selleck products on behavioral activation as locomotor activity was either unaffected, as in the case of ATO infusions in the core and shell, or increased when MPH was infused into either the core and shell sub-region. These findings indicate an apparently ‘opponent’ modulation of premature responses by NE and DA in the NAcb shell or core, respectively, and suggest that the symptom clusters of hyperactive-impulsive type ADHD may have distinct neural and neurochemical substrates. Neuropsychopharmacology (2012) see more 37, 2057-2066; doi:10.1038/npp.2012.53; published online 18 April 2012″
“Biofilms are resilient to a wide variety of environmental stresses. This inherited robustness has been exploited mainly for bioremediation. With a better understanding of their physiology, the application of these living catalysts has been extended to the production

of bulk and fine chemicals as well as towards biofuels, biohydrogen, and electricity production in microbial fuel cells. Numerous challenges call for novel solutions and concepts of analytics, biofilm reactor design, product recovery, and scale-up strategies. In this review, we highlight recent advancements in spatiotemporal biofilm characterization

and new biofilm reactor developments for the production of value-added fine chemicals as well as current challenges and future scenarios.”
“Purpose: In this gmelinol study we identified preoperative or intraoperative factors responsible for the early return of continence after robot-assisted radical prostatectomy using data from a high volume center.

Materials and Methods: Data from 1,299 patients who underwent robot-assisted radical prostatectomy performed by a single surgeon from January 2008 to June 2010 were collected prospectively and analyzed retrospectively. Patients were categorized according to whether they regained continence (no pad and no urinary leakage) within 3 months and variables were then compared. A self-administered validated questionnaire (Expanded Prostate Cancer Index Composite) was used for assessment of continence status and time to recovery.

Results: Within 3 months after surgery 86.3% of patients (1,121/1,299) had recovered continence. Multivariable Cox regression analysis revealed that only age (p < 0.

These findings suggest that allogeneic HSCT may overcome the unfa

These findings suggest that allogeneic HSCT may overcome the unfavorable impact of t(6; 9)(p23; q34) as an independent prognostic factor. Leukemia (2012) 26, 461-464; doi:10.1038/leu.2011.229; published online 26 August 2011″
“Introduction: The sigma(1) ligands are considered to be a new class of potential therapeutic agents for several types of central nervous system check details disorder.

Carbon-11-labeled 1-[2-(3,4-dimethoxyphenyl)ethyl]-4-(3-phenylpropyl)piperazine ([C-11]SA4503) was shown to be a promising PET ligand for mapping sigma(1) receptors, and was applied to measure receptor occupancy with several therapeutic drugs in the living human brain. In this study, we applied this technique for preclinical in vivo screening of novel sigma(1) selective agonists.

Methods: Six newly synthesized piperazine derivatives containing arylalkylamine groups and cyclohexylamine derivatives containing phenyl groups were selected and tested for their in vivo sigma(1) receptor binding with [C-11]SA4503.

The test compounds were administered by intravenous co-injection or oral administration. The in vivo receptor binding of [C-11]SA4503 was evaluated by a tissue dissection method at a single time point.

Results: Our in vivo screen identified the most promising candidate of novel sigma(1) agonist in the piperazine derivatives. Some correlations between in vitro affinity and in vivo receptor blocking rate were observed when considering see more oral bioavailability. In vivo receptor blocking of piperazine derivatives after oral administration may be predictable by simple co-injection study.

Conclusion: Ligand selection

with [C-11]SA4503 by the in vivo receptor binding assay was performed successfully. This technique is a practical and high-throughput method that can directly evaluate blood-brain barrier permeability, receptor binding, and bioavailability of drug candidates at the same time. (C) 2012 Elsevier Inc. All rights reserved.”
“The PcF protein from Phytophthora cactorum is the first member of the “”PcF toxin family”" from the plant PFKL pathogens Phytophthora spp. It is able to induce withering in tomato and strawberry leaves. The lack of sequence similarity with other proteins hampers the identification of the molecular mechanisms responsible for its toxicity. Here, we show that the six cysteines form a disulphide pattern that is exclusive for PcF and essential for the protein withering activity. The NMR solution structure identifies a novel fold among protein effectors: a helix-loop-helix motif. The presence of a negatively charged surface suggests that it might act as a site of electrostatic interaction. Interestingly, a good fold match with Ole e 6, a plant protein with allergenic activity, highlighted the spatial superimposition of a stretch of identical residues. This finding suggests a possible biological activity based on molecular mimicry.


“It has been reported that glucocorticoid (Gc) can induce


“It has been reported that glucocorticoid (Gc) can induce neuronal cell toxicity in the hippocampus. In addition, we examined that serum Gc increased by restraint stress aggravated kainic acid (KA)-induced neuronal death in hippocampal CA3 region. However, the effect of other stressful stimulus like lipopolysaccharide (LPS) increasing serum Gc on KA-induced neuronal death was not elucidated until now. Thus, we examined the time course effect of LPS on KA-induced neuronal death in the hippocampal

CA3 region of mice, especially to address the role of Gc and inflammatory mediators. In the present study, we found that an aggravating effect of LPS on KA-induced neuronal death was correlated with an alteration of

hippocampal IL-1 beta mRNA level at all time points, and the serum Gc and hippocampal IL-1 beta mRNA level was peak at 90 Bcl-2 inhibitor min after LPS treatment (LPS 90 min) when the aggravating effect AZD5582 of LPS on KA-induced neuronal death was maximum. In addition, RU38486 (glucocorticoid receptor antagonist) decreased the hippocampal IL-1 beta mRNA level and abolished the aggravating effect of LPS on KA-induced neuronal death at LPS 90 min and 24 h. In the immunohistochemical study, we found activated and ramified microglia (OX-42) and astrocyte (GFAP) at 24 h after LPS treatment (LPS 24 h) in the hippocampus. These results suggest that Gc itself, cytokines triggered by Gc, or both appears to be involved in the LPS effect depending on LPS pretreatment time. Crown Copyright (C) 2010 Published by Elsevier Ltd on behalf of IBRO. All rights reserved.”
“Pigeons were trained in a forced choice task with four alternatives to categorize arrays consisting

of 1, 3, 5, or 8 dots. Before the pigeons chose a comparison stimulus, they were required to peck each dot sequentially. A single peck to a dot, which was defined as an indicating response, changed the color of the dot so that it was differentiated from those that remained to be counted. The pigeons successfully learned to categorize the numerical arrays and then displayed transfer to novel arrays consisting find more of two, four, six, or seven dots, in a manner according to the order of 1 < 2 < 3 < 4 < 5 < 6 < 7 < 8. Subsequent tests revealed that the pigeons discriminated the stimuli by relying on the number of indicating responses. They also utilized multiple information (surface area, time, and other confounded events), but this was of minor significance, and after training, the pigeons were able to disregard these cues.”
“The gamma isoform of protein kinase C (PKC gamma) is an injury-activated intracellular modulator that boosts neuronal activity in algesic and neuroregenerative signalling pathways. Acetyl-L-carnitine (ALCAR), a physiological compound with role in bioenergetic functions, shows an antihyperalgesic effect and at the same time can exert neuroregenerative and neuroprotective effects.


“A simple method is described for using principal componen


“A simple method is described for using principal component analysis (PCA) to score rat sleep recordings as awake, rapid-eye-movement (REM) sleep, or non-REM (NREM) sleep. PCA was used to reduce the dimensionality of the features extracted from each epoch to three, and the projections were then graphed in a scatterplot where the EPZ004777 supplier clusters were visually apparent. The clusters were then directly manually selected, classifying the entire recording at once. The method was tested in a set of ten 24-h rat sleep electroencephalogram (EEG) and electromyogram (EMG) recordings. Classifications by two human raters performing traditional epoch-by-epoch scoring

were blindly compared with classifications by another two human raters using the new PCA method. Overall inter-rater median percent agreements ranged between 93.7% and 94.9%. Median Cohen’s kappa coefficient ranged from 0.890 to 0.909. The PCA method on average required about

5 min for classification of each 24-h recording. The combination of good accuracy and reduced time compared to traditional sleep scoring suggests that the method may be useful for sleep research. (C) 2009 Elsevier Ireland Ltd. All rights reserved.”
“Human immunodeficiency virus type 1 (HIV-1) vectors transduce rhesus blood cells poorly due to a species-specific block by TRIM5 alpha and APOBEC3G, which target HIV-1 capsid CBL0137 in vivo and viral infectivity factor (Vif), respectively. We sought to develop a lentiviral vector capable of transducing both human and rhesus blood cells by combining components of both HIV-1 and Endodeoxyribonuclease simian immunodeficiency virus (SIV), including SIV capsid (sCA) and SIV Vif. A chimeric HIV-1 vector including sCA (chi HIV) was superior to the conventional

SIV in transducing a human blood cell line and superior to the conventional HIV-1 vector in transducing a rhesus blood cell line. Among human CD34(+) hematopoietic stem cells (HSCs), the chi HIV and HIV-1 vectors showed similar transduction efficiencies; in rhesus CD34(+) HSCs, the chi HIV vector yielded superior transduction rates. In in vivo competitive repopulation experiments with two rhesus macaques, the chi HIV vector demonstrated superior marking levels over the conventional HIV-1 vector in all blood lineages (first rhesus, 15 to 30% versus 1 to 5%; second rhesus, 7 to 15% versus 0.5 to 2%, respectively) 3 to 7 months postinfusion. In summary, we have developed an HIV-1-based lentiviral vector system that should allow comprehensive preclinical testing of HIV-1-based therapeutic vectors in the rhesus macaque model with eventual clinical application.”
“Attention-deficit/hyperactivity disorder (ADHD) in children is characterized by elevated levels of slow wave activity and reduced fast wave activity in resting-state electroencephalogram (EEG). In adults with ADHD, resting-state EEG findings are scarce and inconsistent.

(C) 2012 Elsevier Ltd All rights reserved “
“Objective: Jun

(C) 2012 Elsevier Ltd. All rights reserved.”
“Objective: Junctional ectopic tachycardia is common after pediatric heart surgery. After tetralogy of Fallot repair, the incidence of junctional ectopic tachycardia may be as high as 15% to 20%. We introduced prophylactic

amiodarone for tetralogy repair. This study was conducted to evaluate the effectiveness of the prophylactic amiodarone.

Methods: A continuous infusion of amiodarone was started in the operating room at the time of rewarming during cardiopulmonary bypass at a rate of 2 mg/kg/d and continued for 48 hours. Between November 2005 and November 2009, 63 consecutive patients underwent primary repair of tetralogy, of whom 20 had prophylactic amiodarone (amiodarone group) and 43 did not (control group). Variables studied included demographic and bypass data, surgical procedure details Z-IETD-FMK clinical trial (transannular or nontransannular patch), preoperative and postoperative echocardiography findings, and postoperative inotropic support. Univariate and stepwise multivariate analyses were conducted to determine factors associated with the occurrence of junctional ectopic tachycardia.

Results:

https://www.selleckchem.com/products/incb28060.html The incidence of junctional ectopic tachycardia was 37% in the control group and 10% in the amiodarone group. The groups were similar in age, weight, bypass time, rate of transannular patch usage, and preoperative and postoperative gradient through the right ventricular outflow tract. Prophylactic amiodarone was significantly negatively associated with junctional ectopic tachycardia by both univariate (P = .039) and multivariate (P = .027) analyses. There were no adverse events attributable to prophylactic

amiodarone use.

Conclusions: Prophylactic amiodarone is well tolerated and significantly associated Axenfeld syndrome with a decreased incidence of junctional ectopic tachycardia after tetralogy repair. (J Thorac Cardiovasc Surg 2012;143:152-6)”
“The detection, correlation, and comparison of peptide and product ions from a data independent LC-MS acquisition strategy with data dependent LC-MS/MS is described. The data independent mode of acquisition differs from an LC-MS/MS data acquisition since no ion transmission window is applied with the first mass analyzer prior to collision induced disassociation. Alternating the energy applied to the collision cell, between low and elevated energy, on a scan-to-scan basis, provides accurate mass precursor and associated product ion spectra from every ion above the LOD of the mass spectrometer. The method therefore provides a near 100% duty cycle, with an inherent increase in signal intensity due to the fact that both precursor and product ion data are collected on all isotopes of every charge-state across the entire chromatographic peak width.


“Evolutionary pressure has selected antibodies as key immu


“Evolutionary pressure has selected antibodies as key immune molecules acting against foreign pathogens. The development of monoclonal antibody technology has allowed their widespread use in research, real-time diagnosis and treatment of multiple diseases, including cancer. However, compared with hematologic malignancies, Selleckchem RAD001 solid tumors have often proven to be relatively

resistant to antibody-based therapies. In an attempt to improve the tumor-targeting efficacy of antibodies, new formats with modified, multivalent properties have been generated. Initially, these formats imitated the structure of native IgG, creating mostly monospecific, bivalent antibodies. Recently, novel trivalent antibodies have been developed to maximize tumor targeting capabilities through enhanced biodistribution and functional affinity. We review recent advances in the engineering of multivalent antibodies and further discuss their promise as agents for in vivo diagnostics and therapy.”
“Background: Weight gain is a major Napabucasin solubility dmso side effect of antipsychotic treatment. Some atypical antipsychotic agents have profound effects on weight. Body weight is regulated by a complex system, including both peripheral and central factors. Two of the hormones that seem to play an important role in the regulation of food intake. energy metabolism,

and body weight are leptin and ghrelin. Leptin is a mediator of long-term regulation of energy balance, suppressing food intake and thereby inducing weight loss. Ghrelin on the

other hand is a fast-acting hormone, seemingly playing a role in meal initiation. In this present study it is aimed to compare the effects of five different atypical antipsychotic medications on leptin and ghrelin.

Method: 112 patients who were treated either with clozapine (n = 20), olanzapine (n = 28), risperidone (n = 22), quetiapine (n = 20) or amisulpride (n = 22) as monotherapy for at least one year and age, gender, and body mass index (BMI) matched control group (n=23) were assessed cross-sectionally. Ghrelin and leptin levels were measured with enzyme-immunoassay.

Results: When fasting serum leptin levels were compared between groups, control group had the highest mean value (9.2 +/- Selleck Pazopanib 6.7) and amisulpride group had the lowest mean value (3.7 +/- 2.1) but still there was no statistically significant difference between six groups (F=1993, p=0.084). In the comparison of the mean values of fasting serum ghrelin levels there was a statistically significant difference between groups (F= 11,473, p=0.00). In post-hoc analysis it was seen that the control group had the lowest ghrelin level (194.5 +/- 86.8). Quetiapine treated group (378.1 +/- 260.4) had similar fasting serum ghrelin levels to control group. All the other antipsychotic treatment groups had significantly higher levels of fasting serum ghrelin compared to control group, highest in amisulpride treated group (597.0 +/- 150.0).

Published by Elsevier B V “
“Patients with basal ganglia (BG

Published by Elsevier B.V.”
“Patients with basal ganglia (BG) pathology are consistently found to be impaired on rule-based category learning tasks in which learning is thought to depend upon the use of an explicit, hypothesis-guided strategy. The factors that influence this impairment remain Barasertib unclear. Moreover, it remains unknown if the impairments observed in patients with degenerative disorders such as Parkinson’s disease (PD) are also observed in those with focal BC lesions. In the present study, we tested patients with either focal BC lesions or PD on two categorization tasks that varied in terms of their demands on selective

attention and working memory. Individuals with focal BG lesions were impaired on the task in which working memory demand was high and performed similarly to healthy controls on the task in which Selleckchem A-1210477 selective-attention demand was high. In contrast, individuals with PD were impaired on both tasks, and accuracy rates did not differ between on and off medication states for a subset of patients who were also tested after abstaining from dopaminergic medication. Quantitative, model-based analyses attributed the performance deficit for both groups in the task with high working memory demand to the utilization of suboptimal strategies,

whereas the PD-specific impairment on the task with high selective-attention demand was driven by the inconsistent use of an optimal strategy. These data suggest that the demands on selective attention and working memory affect the presence of impairment in patients with focal BC lesions and the nature of the impairment in patients with PD. (C) 2010 Elsevier Ltd. All rights reserved.”
“A mathematical model was developed to predict the viability of airborne viruses. The model uses water activity as the primary independent variable and an exponential decay function for the viability of the virus. This model was tested using published experimental data obtained by different investigators for influenza. Langat and polio viruses.

The aerosolized media were modelled as a binary solution of water and sodium chloride. The water activity is related directly to the solute concentration in the binary solution. The minimum viability usually occurred just above the efflorescence point, which is the relative humidity at which Flavopiridol (Alvocidib) the solution crystallizes. The relationship between water activity and relative humidity is based on the Kohler theory, whereby the Kelvin term was taken into account. Physical explanations are provided on the variation of viral viability at different relative humidity levels. The predictions obtained by the proposed mathematical model compare well with most of the published experimental data. (C) 2009 Elsevier B.V. All rights reserved.”
“There has recently been a resurgence in the idea that amnesia may be characterized by an increased susceptibility to interference.