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61. Bergsbaken T, Cookson BT: Innate immune response during Yersinia infection: critical modulation of cell death mechanisms through phagocyte activation. J Leukoc Biol 2009, 86:1153–1158.PubMedCrossRef Competing interests The authors declare that they have no competing interests. Authors’ contributions BC participated in the design of the study, conducted experiments, and drafted and finalized the manuscript. KM conducted experiments. IF participated in the design of the study, performed the statistical analysis, and helped draft the manuscript. PL helped draft the manuscript. SM conceived of the study, obtained funding, and Flavopiridol (Alvocidib) participated in its design and coordination and helped to draft the manuscript. All authors read and approved the final manuscript.”
“Background Staphylococcus epidermidis and other coagulase-negative staphylococci (CoNS) constitute the most frequent causes of hospital-acquired infections and are often associated with the use of medical devices [1]. Virulence is mainly attributed to surface colonization and mTOR tumor biofilm formation [2]. A biofilm represents an adherent, structured, high density community of bacterial cells [3] embedded in an extracellular matrix, previously called slime.